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The complete amino acid sequence of alpha-1-microglobulin (alpha1m), the major serum protein, has been determined. The protein is monomeric (Mr=22,000) and is synthesized by hepatocytes as the inactive form, pre-alpha1m, which contains carbohydrate groups. After liberation from the precursor by a plasma protease, it is post-translationally modified to the active form, mature alpha1m, which does not contain carbohydrate and has a molecular weight of 13,000. Pre-alpha1m and mature alpha1m are both covalently linked to a serum transport protein, the major histocompatibility complex-associated protein, class I glycoprotein. The sequence data show that the high abundance of alpha1m in serum is due to a high concentration of pre-alpha1m, the half-life of which is much longer than that of the active protein.Q:
How to delete last n elements in a linked list in JavaScript?
deleteLast2(head, n)
My LinkedList class would have a method that returns the first n elements of a linked list. I am not sure what the best way of deleting the last n elements in my linked list would be. How could I do this?
A:
Your linked list is not a linked list if it doesn’t store a reference to the next node.
To implement the “Delete first n nodes” operation, you just find the pointer to the first node you want to delete and remove it.
A really clean implementation would be to extract a function from this operation to “Delete first n elements and
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Numerous adrenergic nerve terminals were found in the subservient zone of the adrenal cortex. Stimulation with acetylcholine or physostigmine (0.1 mg/kg) reduced the number of these terminals. The results of the experiment with 3H-tyrosine showed that the stimulation of the terminals resulted in alterations of the ultrastructure of the membrane of the nerve fibres. The importance of these nerve terminals for the activity of the adrenal cortex is discussed.Unconventional T cell responses in chronically HIV-1 infected men with progressive disease.
Based on the finding that progression of HIV disease is strongly associated with a poor adaptive immune response, we have initiated a long-term study of chronically HIV-1 infected men to assess the T cell response to HIV proteins. Cell-mediated immune responses to several recombinant (r)HIV proteins and a group of HIV-1 peptides (CTL H-8, H-6, 447-50, 447-69, 734-50, 734-69) were studied. Peripheral blood mononuclear cells (PBMCs) of HIV-1 infected individuals with progressive disease (PPD+; CD4 cell counts
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